www.houseofenoch.com/diseases/multiple-sclerosis/ "Multiple Sclerosis (MS) is likely a biomechanical disease caused by the jaw muscles, cranial bones, and veins of the body. The autoimmune paradigm has produced no effective treatment after 70 years as the prevailing hypothesis on the cause of MS. With the enormous MS drug market now over $20 billion a year, the status quo of MS research serves the interests of pharmaceutical companies, neurologists, and diagnostic imaging providers who live lavishly at the expense of patient health with little incentive to terminate this lucrative source of revenue. Under this new model, MS is caused by the jaw muscles temporarily expanding the cranial cavity, particularly during sustained bruxism, by laterally pulling the temporal bones outwards along the squamosal suture. When the bruxing episode ends, the cranial bones deflect back to their normal positions. This cranial expansion-contraction phenomenon compresses and damages the brain globally resulting in diffuse global brain damage. The focal brain lesions that typify MS are also caused by this mechanic. While the intracranial space is temporarily expanded during bruxing, the drop in intracranial pressure causes the compliant intracranial veins to fill with blood in order to increase intracranial pressure back to equilibrium. When the bruxing episode ends, the cranium contracts but the intracranial venous blood volume is greater than normal owing to the filling period caused by the temporarily expanded cranium. Thus, the large volume of venous blood is mechanically squeezed out of the intracranial space until it comes into contact with the internal jugular vein valves. The obstruction causes the blood to rebound back into the intracranial space, thus mechanically inducing the perivenous focal lesions. Diffuse spinal cord damage and atrophy is caused by Wallerian degeneration from accumulating axonal loss in the brain. Focal spinal cord lesions are caused by venous reflux into the epidural space which shift the position of the spinal cord locally. This mechanical phenomenon stresses the cord’s flanks at the attachment points of the denticulate ligaments, thus causing mechanical damage and creeping fibrosis of the spinal cord’s flanks. Similarly, venous reflux into the lumbosacral veins surrounding the dural sac will displace fluid headwards which drags the spinal cord headwards as well, likewise stressing the denticulate ligament anchorage points. This biomechanical conception of MS is supported by a substantial amount of radiological, histopathological, and epidemiological evidence."